What's New
On October 14, 2007, Governor Schwarzenegger signed into law Assemblywoman
Fiona Ma's Stop Toxic Toys Bill (AB 1108). This new law prohibits the
manufacture, sale, and distribution of children's toys and feeding
products that contain phthalates.
Since our victory last year, California's own Senator Diane Feinstein introduced similar legislation that passed the U.S. Senate in March as part of reform efforts targeted at the Consumer Product Safety Commission.
For tips on how to reduce your child's exposure to toxic chemicals, click here.
How You Can Help
Thank the governor for signing AB 1108 into law.
Report Documents Toxics in Baby Bottles
Environment California Research and Policy
Center released a report, “Toxic Baby Bottles,” showing that a
hormone-disrupting toxic chemical linked to developmental, neural, and
reproductive problems—bisphenol A—leaches from clear, plastic baby bottles into
liquids contained in the bottles. Bisphenol A is linked to numerous adverse
health effects, including early onset puberty, behavioral problems, impaired immune
system, obesity and diabetes, and cancer. Environment California Research
and Policy Center worked with an independent
laboratory to analyze five of the most popular brands of baby bottles on the
market to determine whether bisphenol A leached from the bottles. All five
bottle brands leached bisphenol A at levels founds to cause harm in numerous
laboratory animal studies. More.
For tips on how to protect your children from
exposure to toxic chemicals, click here.
Another Scientific Study Links Chemicals in Kids’ Products to Long-Term
Health Effects
A few years ago, scientists discovered that exposure to
bisphenol A prevents chromosomes from lining up correctly, resulting in
chromosome sorting errors like the kind that cause Down syndrome. While a
variety of possible events could also lead to the same genetic outcome, the
fact that a common chemical can cause this effect is cause for concern.
A recent study shows that exposure to bisphenol A can
lead to chromosomal abnormalities that affect future generations as well.1
This is because female mammals, including mice and humans, form their eggs
while still in their mother’s womb. The study shows that exposure to bisphenol
A by mice during pregnancy disturbs early egg development in the unborn female
fetuses. When these fetuses reach adulthood, the disturbance is translated into
an increase in chromosomally-abnormal eggs and embryos. Chromosome
abnormalities are the largest known cause of spontaneous miscarriage in people.
Thus, eggs that will become a female’s grandchildren are affected through in utero exposure to bisphenol A.
[1] M. Susiarjo, T.J.
Hassold, E. Freeman and P.A. Hunt, “Bisphenol A Exposure In Utero Disrupts Early Oogenesis
in the Mouse,” PLoS Genetics 3(1): e5 doi:10.1371/journal.pgen.0030005, 2007.